Muscles require a supply of oxygen to function but are limited by energetic glycogen resources. In case the intensity of aerobic exercise increases, which requires a faster supply of oxygen than the transport system is able to provide, the exercising muscle tissue switches to the anaerobic extraction of energy.
This leads to the overproduction of lactate and the development of metabolic acidosis. The lactate decreases mobilisation of fat reserves and leads to greater utilisation of glycogen as an energy source. The limiting factor is a critical decrease in glycogen reserves and subsequent hypoglycaemia, especially in CNS. The result is decreased glycolysis with reduced formation of ATP and CP.
Acidosis worsens conditions for the formation and use of muscle potential, thus worsening the contractility of muscles. This leads to physiological muscle fatigue, which is sensed as a heaviness, weakness or pain or stiffness of the skeletal muscles.
Fatigued and exhausted muscles tend to tremble and suffer from spasms. In muscle fatigue, the following insufficiencies occur: decrease in muscle strength, loss of speed and fine motorics.
In repetitive locomotor activities, when breaks are not sufficient for full recovery, acute pathological fatigue occurs, which can end in blood circulation failure and death, or, in chronic pathological fatigue, it can lead to a reduction and atrophy of muscle fibres and their substitution by ligament tissue.
Read the results of the following clinical studies that demonstrate the success rate of low-frequency pulsed magnetic therapy in treatment of this condition.
Related diagnosis: Muscle fatigue